When is the best time to do B-ultrasound for polycystic ovary syndrome?

After polycystic ovary syndrome occurs, an ultrasound examination should be performed. Not only that, patients also need to undergo a laparoscopy. These examination items can determine the real cause of this condition. Generally, after it occurs, patients will have a series of adverse reactions. The most common one is irregular menstruation or anovulation, which should be treated based on the examination results.

(1) Basal body temperature measurement

(2) Ultrasound examination

(3) Diagnostic curettage

(4) Laparoscopy

Hormone measurement

The full name of PCOS is polycystic ovary syndrome, which has menstrual disorders (oligomenorrhea or amenorrhea), anovulation, hirsutism, obesity, infertility, etc. as the main clinical manifestations. It is characterized by bilateral enlargement of the ovaries showing polycystic changes. Its endocrine characteristics are excessive androgen, insulin resistance and hyperinsulinemia, increased LH/FSH ratio and persistent anovulation. It is one of the common causes of menstrual disorders and infertility in women of childbearing age.

1. Promoting fertility For patients who want to have children, the purpose is to promote fertility:

(1) Drug-induced ovulation treatment: Choose appropriate drugs to induce ovulation based on the patient's specific situation. Polycystic ovulation can easily lead to ovarian hyperstimulation syndrome, so patients must follow the doctor's advice when taking medication;

(2) For PCOS patients who are not responsive to drug treatment and still cannot conceive normally, surgical treatment is required. Currently, the best surgical treatment option is minimally invasive ovarian wedge resection, followed by appropriate use of drugs to induce ovulation.

Adjust the menstrual cycle and lower LH. For patients who do not desire to have children, the main purpose is to adjust and restore the menstrual cycle and prevent excessive proliferation of the endometrium or even cancer.

(1) The pituitary gland is more sensitive to GnRH, which leads to excessive secretion of LH, which increases ovarian androgens and promotes follicular atresia. The dominant follicle cannot be formed, and the normal androgen secretion pattern is interrupted. Many small follicles still secrete estrogen. Excessive androgens are converted into estrogen in the periphery. Polycystic ovary syndrome presents with high androgen and high estrogen, with excessive androgen being dominant. (2) Insulin resistance leads to hyperinsulinemia, which stimulates the synthesis and secretion of androgens by theca cells, ovarian stroma, and adrenal cortex.

As androgens increase, estrogen conversion increases. Positive feedback causes LH to increase further, while negative feedback reduces FSH secretion. High LH further increases androgens, continuing a vicious cycle of anovulation. Insulin and estrogen promote follicular development, while androgens promote follicular atresia, resulting in follicular development but maturation disorders, no dominant follicle formation, and anovulation.